Search results for "Axonal loss"

showing 4 items of 4 documents

Longitudinal quantitative MRI assessment of cortical damage in multiple sclerosis: A pilot study

2017

PURPOSE Quantitative MRI (qMRI) allows assessing cortical pathology in multiple sclerosis (MS) on a microstructural level, where cortical damage has been shown to prolong T1 -relaxation time and increase proton density (PD) compared to controls. However, the evolution of these changes in MS over time has not been investigated so far. In this pilot study we used an advanced method for the longitudinal assessment of cortical tissue change in MS patients with qMRI in comparison to cortical atrophy, as derived from conventional MRI. MATERIALS AND METHODS Twelve patients with relapsing-remitting MS underwent 3T T1 /PD-mapping at two timepoints with a mean interval of 12 months. The respective co…

Cortical tissuebusiness.industryMultiple sclerosisAxonal lossmedicine.disease030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineNuclear magnetic resonancemedicine.anatomical_structureGliosisCortex (anatomy)medicineRadiology Nuclear Medicine and imagingStatistical analysismedicine.symptombusinessProton density030217 neurology & neurosurgeryCortical atrophyJournal of Magnetic Resonance Imaging
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Unraveling the T-B tangle in anti-CD20 multiple sclerosis therapy.

2019

Significance Multiple sclerosis (MS) is an inflammatory demyelinating disease of the central nervous system. CD8+ T cells have been strongly implicated in MS pathogenesis, but it is unclear whether myelin is a CD8+ T cell autoantigenic target in MS. This study demonstrated that while myelin-specific CD8+ T cells are present at similar frequencies in untreated MS patients and healthy subjects, the proportion of memory and CD20-expressing myelin-specific CD8+ T cells was increased in MS patients, suggesting prior antigen encounter. This activated phenotype was reversible as the memory and CD20-expressing populations of certain myelin-specific CD8+ T cells were reduced following anti-CD20 trea…

Multiple SclerosisCentral nervous systemAxonal lossDiseaseCD8-Positive T-LymphocytesCD8+ T cellsanti-CD20 therapy03 medical and health sciences0302 clinical medicineImmune systemImmunology and InflammationAntigenmedicineHumansMyelin SheathMultidisciplinarybusiness.industryMultiple sclerosisExperimental autoimmune encephalomyelitisBiological Sciencesmedicine.diseaseAntigens CD20medicine.anatomical_structureImmunizationImmunologybusinessmyelin antigen030217 neurology & neurosurgery030215 immunologyProceedings of the National Academy of Sciences of the United States of America
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Peripheral Neuropathy in the Hypereosinophilic Syndrome: A Case Report

1989

We observed a patient with the hypereosinophilic syndrome that showed as a prominent clinical feature peripheral nerve dysfunction. The neuropathy evolved over 4 months and affected sensory and motor functions. Nerve conduction studies and EMG were compatible with axonal neuropathy. Nerve and muscle biopsies revealed severe axonal degeneration with neurogenic atrophy of muscle. Morphometry of peroneal nerve showed marked axonal loss, more prominent in large myelinated fibers. There was no evidence of vasculitis process. Neuropathy is produced by eosinophil-released substances exerting a neurotoxic effect through direct altered vascular endothelial permeability and local mast cell histamine …

Pathologymedicine.medical_specialtyBiopsyAxonal losschemistry.chemical_compoundEosinophiliaBiopsymedicineHumansAxonmedicine.diagnostic_testHypereosinophilic syndromebusiness.industryPeripheral Nervous System DiseasesMiddle Agedmedicine.diseaseMast cellPeripheral neuropathymedicine.anatomical_structureNeurologychemistryImmunologyFemaleNeurology (clinical)businessVasculitisHistamineEuropean Neurology
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Toxic effects on astrocytes of extracellular vesicles from CSF of multiple sclerosis patients: a pilot in vitro study.

2020

Multiple sclerosis (MS) is an autoimmune and degenerative disorder of the central nervous system (CNS) that causes a progressive loss of motor and cognitive perfor-mances. Moreover, since the earlier phases, axonal loss as well as neuronal degener-ation and a failure of oligodendrocytes to promote myelin repair have been demon-strated. In previous studies, it has been shown that the treatment of rat neuronal primary cultures with serum from MS patients can be toxic for neurons. Here we report a pilot investigation showing that CSF from patients contains extracellular vesicles (EVs) able to induce cell death in rat cultured astrocytes. Although these data are still preliminary, they suggest …

Programmed cell deathPathologymedicine.medical_specialtyMultiple SclerosisDegenerative DisorderCentral nervous systemAxonal lossExtracellular vesiclesPathology and Forensic MedicineMyelinExtracellular VesiclesSettore BIO/10 - BiochimicamedicineAnimalsHumansSettore BIO/06 - Anatomia Comparata E CitologiaNeuronsbusiness.industryMultiple sclerosisRGeneral Medicinemedicine.diseaseRatsmedicine.anatomical_structureAstrocytesToxicityMedicineSettore MED/26 - NeurologiabusinessBiomarkersPolish journal of pathology : official journal of the Polish Society of Pathologists
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